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Exacerbated inflammatory stress from such an infection can stimulate an excess of secretions in the lung, including fibrin compounds and proteinaceous materials, impeding the essential gas exchange for the maintenance of vital organs [30]

However, it is yet unknown how ferroptosis functions in SARS-CoV-2 infected dopaminergic neurons or whether it represents a promising new therapeutic target for COVID-19-related PD therapy

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highly lipophilic and poorly water-soluble