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"Primary Mechanisms of Thymosin 4 Repair Activity in Dry Eye Disorders and Other Tissue Injuries." Investigative Ophthalmology & Visual Science
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Lee, E., & Burgess, K
Either Cd38 gene deletion or NAD supplementation successfully reinstated the alterations caused by Ang II stimulation and inhibited sEV packaging of mitochondrial compounds and ubiquitinated proteins, suggesting that CD38 deficiency promoted lysosome-mediated degradation
