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To demonstrate causality between B-017 and mitochondrial-dependent necrotic and apoptotic pathways in ischaemia/reperfusion injury, we first exposed wild-type C57BL/6 and Bnip3 / knockout mice to 30 min ischaemia, followed by 24 h reperfusion

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Evaluation of CysOx reactivity and fluorescence in complex biological settings Encouraged by our success in the CSA model, we then moved on to more targets with greater biological relevance and complexity

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