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Supporting the notion that TERT and TERC deficiency might contribute to pulmonary fibrosis by mechanisms dependent on telomerase activity but not necessarily telomere length, telomerase activity is induced in IPF and NSIP fibroblasts and systemic sclerosis lung compared to healthy donor samples (Fridlender et al., 2007

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It was demonstrated that phosphorylation at the Ser637 site stimulates Ser616 phosphorylation and that only the inhibition of downstream Ser616 phosphorylation results in mitochondrial elongation (Gonzalez et al., 2021)