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PRELIMINARY RESULTS OF A PHASE I/II STUDY OF AZACITIDINE, VENETOCLAX AND PEVONEDISTAT IN PATIENTS WITH SECONDARY ACUTE MYELOID LEUKEMIA WHO ARE UNFIT FOR INTENSIVE CHEMOTHERAPY

41 Slc3a2-dependent BCAA metabolism also has a key role in the physiological activities of Foxp3 + Treg cells, and either BCAA deprivation or SLC3A2 mutation can induce impaired activation of the mTORC1 pathway in Treg cells and inhibit the immunosuppressive function of Treg cells

Those with sensitive digestive systems might need to start with smaller amounts

Mitochondrial dysfunction represents a significant consequence of the immunosuppressive tumor microenvironment on T cells, impeding the supply of acetyl-CoA derived from lipids for the tricarboxylic acid cycle [55, 56]