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Nature 604:697707 Duan K, Premi E, Pilotto A, Cristillo V, Benussi A, Libri I, Giunta M, Bockholt HJ, Liu J, Campora R, Pezzini A, Gasparotti R, Magoni M, Padovani A, Calhoun VD (2021) Alterations of frontal-temporal gray matter volume associate with clinical measures of older adults with Covid-19

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[DOI] [PMC free article] [PubMed] [Google Scholar] 250.Chang C.L., Weigel A.V., Ioannou M.S., Pasolli H.A., Xu C.S., Peale D.R., Shtengel G., Freeman M., Hess H.F., Blackstone C., et al

As stated above, according to several evidence, acute COVID-19 damage to the CNS may play a central role in the pathogenesis of neuropsychiatric symptoms through several pathophysiological mechanisms: dysregulation of inflammation, astrocytic glial damage, glutamatergic impairment (shift in tryptophan metabolism towards kynurenine pathway, Nf-B, oxidative stress, glutamatergic and excitotoxic damage) and possible glymphatic impairment [15, 46, 82]