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These results are consistent with those of a previous study in a cyclophosphamide-induced hepatotoxic model, which reported that PPAR signaling, but not PPAR signaling, mediated antioxidant and anti-inflammatory effects in the liver (19)
This effect was only found with APOE2 and not APOE3 indicating a neuroprotective function for APOE2 [95]
If both answers are yes, there's a reasonable case for it
Baseline to post-intervention changes were not performed for the DCM-T group since only two dogs in this group survived to the 9-month re-evaluation time point
