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Behavioral deficit, oxidative stress, and mitochondrial dysfunction precede tau pathology in P301S transgenic mice
doi: 10.1161/01.STR.0000157663.43209.a2
doi:10.1093/icb/42.3.517 [DOI] [PubMed] [Google Scholar] 113.Enwonwu CO, Sawiris P, Chanaud N
Originally designed to inhibit ALK phosphorylation by interacting with the Y1230 residue and causing G1/S-phase cell cycle arrest and apoptosis [106], Crizotinib also has potent activity against c-MET with exon 14 alterations
