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Description
Their fundamental structural differences explain how these compounds function differently in the body
doi: 10.1136/jnnp.2007.131045
Myricetin mitigated sevoflurane-induced cognitive dysfunction in aged-mice through inhibiting histone deacetylase 2/nuclear factor erythroid 2-related factor 2/heme oxygenase-1 signalling-mediated ferroptosis and mitochondrial dysfunction
By modulating the host immune response, HDT ameliorates immunopathological damage and improves overall outcomes by promoting autophagy, antimicrobial peptide production, and other mechanisms
