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A study found that TECs experience complex molecular and cellular events that are particularly relevant to IRI, including oxidative damage and activation of the innate immune system through Toll-like receptors (TLRs), sphingosine-1-phosphate (S1P) receptors, and hypoxia-inducible factors (HIFs)

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This makes thymosin beta-4 particularly relevant to fracture healing because it directly supports the cellular mobility required at every stage of bone repair