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While these experiments uncovered more information about cellular changes due to deletions of copies of the S-Adenosylmethionine synthetase genes, SAM1 and SAM2 , additional work was needed to try to identify how these mutations, and the resultant alterations in AdoMet concentration, led to the various differences that were observed

KTKEGV repeat motifs are key mediators of normal -synuclein tetramerization: their mutation causes excess monomers and neurotoxicity

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Therefore, we performed pharmacokinetic studies of BPC157 in beagle dogs following single IV administration at a dose of 6 g/kg, single IM administration at doses of 6, 30, or 150 g/kg, and repeated IM administration at a dose of 30 g/kg for seven consecutive days