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CRF in the paraventricular nucleus mediates gastric and colonic motor response to restraint stress

The specific mechanism may also be by activating VEGF-A / VEGFR1 -mediated AKT and p38/MAPK signaling pathways to upregulate downstream eNOS expression, which may increase the NO product synthesis, thus promote angiogenesis of gastric mucosa and also interact with endoplasmic reticulum (ER) stress

Arch Biochem Biophys 615 , 2234, (2017)
