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Glucocorticoid protects rodent hearts from ischemia/reperfusion injury by activating lipocalin-type prostaglandin D synthase-derived PGD2 biosynthesis

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These molecules prevent cumulative cellular damage associated with chronic oxidative stress, which contributes to inflammatory, neurodegenerative processes, premature aging and a range of chronic diseases [31], non-communicable diseases such as cardiovascular, neurodegenerative, certain types of cancer and conditions related to premature ageing [14, 32]

In the latter organ, CRH alone or in combination with AVP stimulates corticotropic cells to release proopiomelanocortin (POMC), which is an ACTH precursor