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Role of mTORC1 in the sensing of defective mitochondria mTORC1 contributes to the detection of defective mitochondria not only by regulating organelle dynamics and isolating damaged organelles through fission but also by modulating communication with the nucleus via retrograde signaling, particularly when the mitochondrial genome needs to be repaired (Fig

This may leave p53 unbound, so that it may target senescent cells for apoptosis

Furthermore, our group has discovered novel oxidation hotspots, including His10 and Leu17 in the B-chain, and Cys6 in the alpha-chain (87)

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