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In a model where the pathway is simplified in on-off states, the transcriptional co-regulator -catenin is continually targeted for proteasomal degradation by the GSK3/APC/Axin complex when Wnt ligands are absent and the pathway is inactive

Subsequently, we will review the existing modifications in PCa tumor immunotherapy that have entered clinical trials but have yet to achieve substantial breakthroughs, along with the potential improvements peptides can offer in this regard

Dysregulation of TLR signaling, stimulated by gut dysbiosis and subsequent local inflammation, may trigger -synuclein aggregation