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With prolonged exposure or high doses, it transitions to a Phase II block, involving both pre- and post-synaptic effects, including impaired acetylcholine release (autoreceptor inhibition) and open-channel block

We can see that a high concentration of H 2 O 2 has a significant impact on GSH and GSSG levels as the GSH is oxidised to GSSG in order to reduce (scavenge) H 2 O 2

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