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In AD patients, HO-1 expression is significantly elevated in reactive astrocytes in the hippocampus and cerebral cortex (Schipper et al., 2009)

While wild-type BPC157 and other alanine-substituted peptides significantly enhanced proliferation, the P3A mutant completely lost this activity (Fig

29 In the study of human MDD transcriptome, there are defects in the transcription levels of somatostatin (SST) in the subgenus anterior cingulate cortex and amygdala of MDD patients, 30,31 and SST levels are directly involved in the cellular processes that affect the synaptic output of intermediate neuronal circuits