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In the U.S., CVD is the leading cause of death, constitutes 17% of healthcare costs, and is estimated to increase in prevalence by ~10% and direct costs by almost three-fold over the next 20 years under current CVD prevention and treatment regimes [2]
In this Review, we focus on the role of the melanocortin 4 receptor and provide an integrative view of the molecular mechanisms that lead to melanocortin-induced changes in synaptic plasticity within these diverse physiological systems
O'Cuinn, CRC Press, New York, 19 und 99101 (1995)
Intracellular toxic advanced glycation end-products in 1.4E7 cell line induce death with reduction of microtubule-associated protein 1 light chain 3 and p62
