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Elucidation of the mechanism of incorporation of insulin in controlled release systems based on complexation polymers
found that XBF promoted mitochondrial fusion by upregulating MFN1 to inhibit NF-B pathways, and subsequently inhibited pro-inflammatory macrophage polarization (161)
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This is particularly relevant for critical-dose drugs such as anticoagulants, antiepileptics, and immunosuppressants
