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The mechanical activation of ENaC is, in part, predicated on N-glycosylation of asparagines, which are thought to act as anchoring points for extracellular tethers that interact with the extracellular matrix and/or the glycocalyx (16, 137)

NRF2 also interacts with oncogenic signaling pathways frequently deregulated in HCC, such as PI3K/AKT, mTOR, and NOTCH1

These vectors provided reasonable levels of -globin transgene expression in vitro , but the transduction of mouse hematopoietic stem cells (HSCs) was suboptimal, resulting in limited and variable expression of the -globin gene in vivo [133], [134]

Cheng T, Sudderth J, Yang C, Mullen AR, Jin ES, Mats JM, DeBerardinis RJ