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An increased activity of estrogen receptors (ERs), a local estrogen production in endometriotic cells, and a progesterone receptor decreased activity (progesterone-resistance) are considered the main causes of impaired apoptosis, reduced immune function, and increased inflammation [18]
Oxytosis/ferroptosis-(Re-) emerging roles for oxidative stress-dependent non-apoptotic cell death in diseases of the central nervous system
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