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We hypothesize that the drastic inhibition of GLP-1 secretion via the shutdown of the main pathway through the SSTRs-Gi axis is the primary cause of pasireotide-induced hyperglycemia

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In this static simulated human gastrointestinal system, commercial PET MPs (50 15 m) were applied at 80 mg/L and sequentially incubated in simulated salivary, gastric, and intestinal fluids