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doi: 10.1210/jc.2007-0606

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5 Besides numerous clinical trials in progress and over dozens of molecules being engineered against VEGF/VEGF receptor (VEGFR) and non-VEGF pathways, 4, 6 the entire situation warrants the understanding of molecular underpinnings and loopholes supporting the bypass angiogenic mechanisms ensuring tumor progression and metastasis even after the treatment with effective anti-angiogenic agents