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Description
Recent studies have shown that drugs such as metformin, itraconazole and pimobendan have been repositioned to regulate metabolic and epithelial-mesenchymal transition (EMT) pathways, effectively inhibiting BC progression (Kandasamy et al., 2024)
These discrepancies underscore the challenges of modelling human genetic variation in experimental systems and emphasise the importance of context-specific follow-up [10]
Notably, Cys119 and Cys162 have been identified as the principal sites for oxidation and significantly influence the regulatory dynamics of MKK3
Sigal S
