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Differential effects of glutathione S-transferase pi (GSTP1) haplotypes on cell proliferation and apoptosis
Agerelated and diseaserelated declines in glutathione have been documented in multiple tissues, which is one reason it is targeted in many wellnessoriented IV protocols, although optimal dosing, frequency, and longterm safety still require individualized medical judgment
However, this does not explain the fact that, in the ER, the ratio of reduced to oxidized glutathione ([GSH]:[GSSG]) is optimal for disulphidebond formation (Lyles & Gilbert, 1991)
