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In the recent Rome Proposal, a COPD exacerbation was described as an event characterised by an acute burst of airways inflammation due to stimuli (e.g., bacteria, viruses, or environmental pollutants) that is coupled with worsening of existing airflow limitation, leading to worsening symptoms (dyspnoea and/or cough and sputum), which may be accompanied by tachypnea and/or tachycardia [4]

doi: 10.3390/molecules30030654 144

In the course of this cholestatic model of liver injury[25], BPC 157 also counteracted all characteristic structural, biochemical, and hemodynamic disturbances, with portal hypertension either not developed, portal pressure remaining normal (BPC 157 prophylactic regimen) or portal hypertension rapidly reversed (delayed post-treatment)