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These proteins are acknowledged as binary molecular adjustments that switch the intracellular signaling pathways [54]

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Overall, the results indicate that PARK2 knockout mice display impaired muscle strength and non-motor behavior and that downregulation of CPT1 activity ameliorates these symptoms, decreases hyperglycemia and diminishes oxidative stress, reactive microglia markers and inflammation in the midbrain based on normalized gene expression and protein concentrations

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