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Description
Changes in phosphatidylcholine metabolism are part of an adaptive program activated in response to stress conditions that contribute to cancer therapy resistance and open therapeutic opportunities for treating drug-resistant cancers
The defects in LMP-1 localization could result from GLO-3 and CCZ-1 functioning in the AP-3 pathway independently of regulating GLO-1
Genotypic distribution and epidemiology of HCV infection in Western Europe countries has changed over the last decades

The carnitine system and cancer metabolic plasticity