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Association and dissociation phases were recorded, and after each cycle the surface was regenerated with 10 mM glycine-HCl (pH 2.0) for 30 s to remove bound analyte

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Transdermal administration of Cu (II)-atsm also significantly extended survival and decreased the percentage of copper-deficient mutant Sod1 in the spinal cord (Williams et al., 2016), suggesting that the improved neurological phenotype may be related to increased Sod1 function

indicated that Dex enhanced mitochondrial function, reduced ROS production, and protected against myocardial damage by upregulating SLC7A11 (cystine transporter solute carrier family 7 member 11) and GPX4 (glutathione peroxidase 4), thereby inhibiting ferroptosis [46]
