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Virus-inducible IGFALS facilitates innate immune responses by mediating IRAK1 and TRAF6 activation
This finding agrees with the literature whereby the role of quinone redox metabolism is an ambiguous source of toxicity, with evidence suggesting that redox cycling requires supra-pharmacological concentrations of doxorubicin to generate substantial ROS in tissues and cells 10
Smad3 deficiency attenuates bleomycin-induced pulmonary fibrosis in mice
