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(2020) 9:R10211
10.1093/cid/ciw088 135 TimminsG
However, larger studies showed no improvements in clinical outcomes (Treeprasertsuk et al., 2003
The distinct roles of GluN2A and GluN2B in neuropsychiatric disorders may be attributed to the properties of GluN2 subunits in determining the receptor deactivation kinetics, channel gating, Ca 2+ permeability, and voltage-dependent blockade by extracellular Mg 2+ [6]
